Physiology and pathophysiology of the islets of Langerhans

Proceedings of the November 1994 meeting. The 59 contributions cover glucose recognition, the role for K (ATP) channels, intracellular messengers, modulation of islet release, beta-cell metabolism and Ca2+, beta-cell heterogeneity: building a homogeneous response from a heterogeneous population, ISSM (insulin dependent diabetes mellitus): mechanisms of beta-cell destruction and defense, pathophysiology of the NIDDM, and islet growth, regeneration, and transplantation. All chapters contain experimental facts and observations that will be of help to those interested in the physiology and pathophysiology of the islet of Langerhans.